Group - Pulmonary edema

, viruses or fungi. Though treatment protocols have significantly advanced since...

, viruses or fungi. Though treatment protocols have significantly advanced since...

comprehensive pulmonary hypertension program, which is considered one of the bes...

About the Video: Margaret Martins suffered from a life-threatening form of pulmo...

About the Video: Pulmonary thromboendarterectomy, or PTE, is a delicate and comp...

Department of Pulmonary medicine,vss medical college,burla. In medicine, pulmono...

In this short video, viewers are introduced to Avelino Verceles, MD, a pulmonary...

Overview: Cardiovascular disease is the leading cause of death in women, just as...

Overview: Cardiovascular disease is the leading cause of death in women, just as...

In this short video, viewers are introduced to Montserrat Diaz-Abad, MD, a pulmo...

In medicine, flash pulmonary edema (FPE), is rapid onset pulmonary edema. It is most often precipitated by acute myocardial infarction or mitral regurgitation, but can be caused by aortic regurgitation, heart failure, or almost any cause of elevated left ventricular filling pressures.

High altitude pulmonary edema (HAPE) is a life-threatening form of non-cardiogenic pulmonary edema that occurs in otherwise healthy mountaineers at altitudes above 2,500 meters (8,200 feet). Some cases however have been reported also at lower altitudes (between 1500 and 2500 m in highly vulnerable subjects), although what makes some people susceptible to HAPE is not currently known. HAPE remains the major cause of death related to high altitude exposure with a high mortality in absence of emergency treatment. The initial insult that causes HAPE is a shortage of oxygen which is caused by the lower air pressure at high altitudes. The mechani...

High altitude pulmonary edema (HAPE) is a life-threatening form of non-cardiogenic pulmonary edema that occurs in otherwise healthy mountaineers at altitudes above 2,500 meters (8,200 feet). Some cases however have been reported also at lower altitudes (between 1500 and 2500 m in highly vulnerable subjects), although what makes some people susceptible to HAPE is not currently known. HAPE remains the major cause of death related to high altitude exposure with a high mortality in absence of emergency treatment. The initial insult that causes HAPE is a shortage of oxygen which is caused by the lower air pressure at high altitudes. The mechani...

High altitude pulmonary edema (HAPE) is a life-threatening form of non-cardiogenic pulmonary edema that occurs in otherwise healthy mountaineers at altitudes above 2,500 meters (8,200 feet). Some cases however have been reported also at lower altitudes (between 1500 and 2500 m in highly vulnerable subjects), although what makes some people susceptible to HAPE is not currently known. HAPE remains the major cause of death related to high altitude exposure with a high mortality in absence of emergency treatment. The initial insult that causes HAPE is a shortage of oxygen which is caused by the lower air pressure at high altitudes. The mechani...

High altitude pulmonary edema (HAPE) is a life-threatening form of non-cardiogenic pulmonary edema that occurs in otherwise healthy mountaineers at altitudes above .

High altitude pulmonary edema (HAPE) is a life-threatening form of non-cardiogenic pulmonary edema that occurs in otherwise healthy mountaineers at altitudes above 2,500 meters (8,200 feet). Some cases however have been reported also at lower altitudes (between 1500 and 2500 m in highly vulnerable subjects), although what makes some people susceptible to HAPE is not currently known. HAPE remains the major cause of death related to high altitude exposure with a high mortality in absence of emergency treatment. The initial insult that causes HAPE is a shortage of oxygen which is caused by the lower air pressure at high altitudes. The mechani...

Edema (American English) or oedema (British English; both words from the Greek d µa), formerly known as dropsy or hydropsy, is an abnormal accumulation of fluid beneath the skin, or in one or more cavities of the body. Generally, the amount of interstitial fluid is determined by the balance of fluid homeostasis, and increased secretion of fluid into the interstitium or impaired removal of this fluid may cause edema. Five factors can contribute to the formation of edema: Generation of interstitial fluid is regulated by the forces of the Starling equation. Hydrostatic pressure within blood vessels tends to cause water to filter out into the...

Abstract: Department of Internal Medicine, Tokyo National Chest Hospital, Japan.

Abstract: Department of Internal Medicine, Tokyo National Chest Hospital, Japan.

Abstract: Omega-oxidized leukotriene B4 detected in the broncho-alveolar lavage fluid of patients with non-cardiogenic pulmonary edema, but not in those with cardiogenic edema. Seeger W, Grimminger F, Barden M, Becker G, Lohmeyer J, Heinrich D, Lasch HG. Intensive Care Unit, Department of Internal Medicine, Giessen, FRG. Leukotriene (LT) generation has been implicated in the pathogenesis of the acute respiratory distress syndrome, ARDS. In the present study, we analysed broncho-alveolar lavage fluids of patients on mechanical ventilation because of ARDS (17 samples taken from 9 patients) or because o...

Abstract: Department of Medicine, Cl.E. Cavell, Brussels, Belgium.

Abstract: Department of Medicine, Cl.E. Cavell, Brussels, Belgium.

Abstract: Responses of pulmonary C-fibre and rapidly adapting receptor afferents to pulmonary congestion and edema in dogs. Ravi K, Kappagoda CT. Division of Cardiology, University of Alberta, Edmonton, Canada. The effects of cardiogenic and noncardiogenic pulmonary edema on the activities of rapidly adapting receptors (RARs) and pulmonary C-fibre receptors were investigated in dogs anaesthetized with chloralose. Cardiogenic pulmonary edema was produced by elevating the mean left atrial pressure by 25 mmHg (1 mmHg = 133.32 Pa) above the control value for a period of 45 min, by partial obstruction of ...

Abstract: Postoperative unilateral pulmonary edema: possible amiodarone pulmonary toxicity. Herndon JC, Cook AO, Ramsay MA, Swygert TH, Capehart J. Department of Anesthesia, Baylor University Medical Center, Dallas, Texas 75246. PMID: 1736706 [PubMed - indexed for MEDLINE]

Abstract: The authors present two cases of unsuccessful hanging which hat lead to a very severe neurological picture and especially the very rapid appearance of acute pulmonary edema resulting in a vertiable flooding of the alveoli. The victims, aged respectively 21 and 15 years, were free from any pre-existing cardiopathy. These cases of pulmonary edema, resistant to depletive therapy (bleeding, diuretics) and cardiotonic agents (Cedilanid) should only be jugulated after a one hour session of oxygen at 3 ATA with a baro-assisted respirator. The following are discussed with reference to these two cases:...

Abstract: Pulmonary edema in pregnancy. Witry SW. University of Virginia Health Sciences Center, Charlottesville 22908. Normal cardiovascular and respiratory changes in pregnancy can predispose women to the development of pulmonary edema. Conditions and treatments unique to pregnancy, such as multiple gestation or tocolysis, further increase this risk. Recognition of risk factors and signs and symptoms of pulmonary edema allows the nurse to intervene quickly, thus decreasing potential complications to the mother and child. PMID: 1640274 [PubMed - indexed for MEDLINE]

Abstract: [Neurogenic pulmonary edema] [Article in Danish] Staach LJ, Schultz A, Knudsen F. Anaestesiologisk/intensiv afdeling, Aalborg Sygehus Syd. Neurogenic pulmonary oedema (NPE) is a recognized, serious, relatively uncommon, acute complication of cerebral insults of various types. Despite numerous animal experimental investigations and case reports, the pathological physiology is not yet elucidated. An acute cerebral insult with raised intracranial pressure is accompanied by violent activation of the sympathetic system and increase in the concentrations of circulating catecholamines. Probab...