Group - Pulmonary edema

, viruses or fungi. Though treatment protocols have significantly advanced since...

, viruses or fungi. Though treatment protocols have significantly advanced since...

Chronic obstructive pulmonary disease affects 12 million Americans—most of the...

More than 40000 people die each year from idiopathic pulmonary fibrosis, a condi...

About the Video: Margaret Martins suffered from a life-threatening form of pulmo...

comprehensive pulmonary hypertension program, which is considered one of the bes...

About the Video: Pulmonary thromboendarterectomy, or PTE, is a delicate and comp...

In this short video, viewers are introduced to Montserrat Diaz-Abad, MD, a pulmo...

Department of Pulmonary medicine,vss medical college,burla. In medicine, pulmono...

July 25, 2007 presentation by Norman Rizk for the Stanford School of Medicine Me...

In medicine, flash pulmonary edema (FPE), is rapid onset pulmonary edema. It is most often precipitated by acute myocardial infarction or mitral regurgitation, but can be caused by aortic regurgitation, heart failure, or almost any cause of elevated left ventricular filling pressures.

High altitude pulmonary edema (HAPE) is a life-threatening form of non-cardiogenic pulmonary edema that occurs in otherwise healthy mountaineers at altitudes above 2,500 meters (8,200 feet). Some cases however have been reported also at lower altitudes (between 1500 and 2500 m in highly vulnerable subjects), although what makes some people susceptible to HAPE is not currently known. HAPE remains the major cause of death related to high altitude exposure with a high mortality in absence of emergency treatment. The initial insult that causes HAPE is a shortage of oxygen which is caused by the lower air pressure at high altitudes. The mechani...

High altitude pulmonary edema (HAPE) is a life-threatening form of non-cardiogenic pulmonary edema that occurs in otherwise healthy mountaineers at altitudes above 2,500 meters (8,200 feet). Some cases however have been reported also at lower altitudes (between 1500 and 2500 m in highly vulnerable subjects), although what makes some people susceptible to HAPE is not currently known. HAPE remains the major cause of death related to high altitude exposure with a high mortality in absence of emergency treatment. The initial insult that causes HAPE is a shortage of oxygen which is caused by the lower air pressure at high altitudes. The mechani...

High altitude pulmonary edema (HAPE) is a life-threatening form of non-cardiogenic pulmonary edema that occurs in otherwise healthy mountaineers at altitudes above 2,500 meters (8,200 feet). Some cases however have been reported also at lower altitudes (between 1500 and 2500 m in highly vulnerable subjects), although what makes some people susceptible to HAPE is not currently known. HAPE remains the major cause of death related to high altitude exposure with a high mortality in absence of emergency treatment. The initial insult that causes HAPE is a shortage of oxygen which is caused by the lower air pressure at high altitudes. The mechani...

High altitude pulmonary edema (HAPE) is a life-threatening form of non-cardiogenic pulmonary edema that occurs in otherwise healthy mountaineers at altitudes above .

High altitude pulmonary edema (HAPE) is a life-threatening form of non-cardiogenic pulmonary edema that occurs in otherwise healthy mountaineers at altitudes above 2,500 meters (8,200 feet). Some cases however have been reported also at lower altitudes (between 1500 and 2500 m in highly vulnerable subjects), although what makes some people susceptible to HAPE is not currently known. HAPE remains the major cause of death related to high altitude exposure with a high mortality in absence of emergency treatment. The initial insult that causes HAPE is a shortage of oxygen which is caused by the lower air pressure at high altitudes. The mechani...

Edema (American English) or oedema (British English; both words from the Greek d µa), formerly known as dropsy or hydropsy, is an abnormal accumulation of fluid beneath the skin, or in one or more cavities of the body. Generally, the amount of interstitial fluid is determined by the balance of fluid homeostasis, and increased secretion of fluid into the interstitium or impaired removal of this fluid may cause edema. Five factors can contribute to the formation of edema: Generation of interstitial fluid is regulated by the forces of the Starling equation. Hydrostatic pressure within blood vessels tends to cause water to filter out into the...

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Abstract: High altitude constitutes an exciting natural laboratory for medical research. Over the past decade, it has become clear that the results of high-altitude research may have important implications not only for the understanding of diseases in the millions of people living permanently at high altitude, but also for the treatment of hypoxemia-related disease states in patients living at low altitude. High-altitude pulmonary edema (HAPE) is a life-threatening condition occurring in predisposed, but otherwise healthy subjects, and, therefore, allows to study underlying mechanisms of pulmonary edema...

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Abstract: Serum and pulmonary edema fluid samples of 26 patients with pulmonary edema were examined. The correlation coefficient comparing measured colloid osmotic pressure (COPm) to calculated colloid osmotic pressure (COPc) (Landis and Pappenheimer equation) was 0.84. Significant differences between COPm and COPc were noted when total protein (TP) concentrations were less than or greater than 5 g/dl (p < 0.001 and p < 0.01, respectively). Twenty-one of 69 samples (30%) had a greater than 4 mm Hg difference between measured and calculated values. COP should be measured rather than calculated for accura...

Abstract: This case report describes a 61-year-old man who developed reexpansion pulmonary edema (RPE) of the collapsed left lung after video-assisted thoracoscopic surgery because of left thoracic empyema, complicated with secondary contralateral pulmonary edema later. The left lung was gently reexpanded after surgery under one-lung ventilation anesthesia for 2.5 hours. The patient developed RPE of the left lung immediately after surgery, and required mechanical ventilation with positive end-expiratory pressure support. RPE was resolved within 24 hours. Nevertheless, delayed onset of contralateral pulm...

Abstract: We experienced a case of reexpansion pulmonary edema (RPE) after surgical treatment of pneumothorax. In this case, protein leakage and polymorphonuclear leukocyte (PMN) accumulation were observed in the reexpanded lung. Interleukin-8 and leukotriene B4 in edema fluid were increased at the onset of RPE. PMN elastase was also increased, though its peak was delayed. The plasma level of P-selectin, which mediates adhesion between PMN and endothelium, was elevated. We speculate that some of these fluid mediators may play important roles in chemotaxis and activation of PMN in the development of RPE.

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Abstract: BACKGROUND: Congestive heart failure is a common cardiac disorder associated with a high mortality. There are a limited number of prognostic scales predicting in-hospital outcomes after an acute episode of congestive heart failure. OBJECTIVES: The goal of this investigation was to develop a simple prognostic score predicting in-hospital outcome in patients with acute cardiogenic pulmonary edema. METHODS: We retrospectively studied 276 consecutive patients hospitalized with acute pulmonary edema from the years 1998 to 2000. RESULTS: During the initial hospitalization, 58 patients (21%) died and...

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